Overcoming Weight-Loss Inequity
Author's link at: amazon.com/author/crhornbeckkaiser
Weight management inequity tends to exasperate many seeking to lose weight.
I. A Clearly Visible Problem
We look around and see skinny people (less of them as the growing overweight and obesity epidemic envelopes two thirds of adults in America). More vexing still, many who seem thin also embrace terrible dietary practices. They seem to be able to down copious quantities of the worst high glycemic junk foods with impunity.
Whether we say it or not, a part of us demands, "Where is the justice in this?"
II. A Misunderstood Cause
As presented in 1 to 5 Weight-Loss without Dieting, insulin resistance is the culprit in this apparent inequity.
Our metabolisms become overwhelmed by a constant barrage of carbohydrates (the American dietary trend is one of ever increasing high carb, high glycemic binging).
The mechanism is explained clearly in 1 to 5 Weight-Loss without Dieting, but the short of it is that our cells become resistant to the ongoing barrage of insulin produced by our bodies to transport the glucose that results from eating carbohydrates.
Still, some will ask, "Why do thin members of our population appear able to withstand this high carb barrage without gaining weight?"
III. A Genetic Basis for Aging
It would appear that the cells of these individuals are able to handle the inundation of insulin that results from a high carb diet.
The reason is likely to be found on the genetic level.
Just as the Hayflick Limit -- described in Chapter Fourteen of 1 to 5 Weight-Loss without Dieting -- dictates the genetic rate of aging, so it is suspected (by me, at least!) that a genetic operator controls our cellular ability to tolerate the insulin barrage of ongoing high carb eating.
A parallel may be seen in the work of Nobel Laureate Elizabeth Blackburn. She discovered that the "ends" of chromosomes (telomeres) are shortened in cell replication. This continues until a "terminal" shortening resulting in cell death occurs.
This process explains the results of aging that we all experience.
IV. A Parallel Genetic Basis for Insulin Resistance
What if a similar genetic process operates with regard to carbohydrate metabolism?
What if a genetic receptor that controls cell ability to process insulin is "shortened" at genetically determined rates?
Insulin resistance would then be a result of a combination of insulin exposure by dietary practices AND by genetic inheritance (that predetermines our ability to tolerate an ongoing carbohydrate mediated insulin barrage).
If this is the case -- and I suspect it is -- then it should be possible to identify a genetic means by which insulin "shortened" genes could be restored.
When this is discovered, excess high carb eating would not have to produce the epidemic of obesity threatening and destroying the health of untold millions.
V. A Wonderful Solution
Until then, those of us who have embraced the 1 to 5 Weight-Loss without Dieting approach feast up to eight times per day on the most amazingly delicious and healthful low-carb, high healthful fat, high protein foods imaginable!
- I've never eaten better, never eaten more and never enjoyed eating as much as I do now! I weigh what I weighed in high school (about 176 pounds), enjoy increasing participation in mountain biking, tennis and weight lifting, wear slim clothes (which I think make me look better!) and have more energy and acuity than ever!
If this sounds good to you, please check out the Kindle or print edition of 1 to 5 Weight-Loss without Dieting. (The Kindle edition is readable on cell phones and on computers per Amazon's quick and free Kindle app download!)
You owe it to yourself to read this book!
Author's link at: amazon.com/author/crhornbeckkaiser
